Delos Santos KA, Yergeau C, Moon N-S (2018) Patient-derived phosphoribosyl pyrophosphate synthetase mutations in Drosophila result in autophagy and lysosome dysfunction. bioRxiv
Kim M, Tang JP, Moon N-S (2018) An alternatively spliced form affecting the Marked Box domain of Drosophila E2F1 is required for proper cell cycle regulation. PLoS Genetics 14(2): e1007204.
Bradley-Gill MR, Kim M, Feingold D, Yergeau C, Houde J, Moon NS (2016). Alternate transcripts of the Drosophila “activator” E2F are necessary for maintenance of cell cycle exit during development. Developmental Biology 411(2):195-206
Collins H, Moon NS (2013) The components of Drosophila histone chaperone dCAF-1 are required for the cell death phenotype associated with rbf1 mutation. G3: Genes | Genomes | Genetics doi:10.1534/g3.113.007419.
Longworth MS, Walker JA, Anderssen E, Moon NS, Gladden A, Heck MM, Ramaswamy S, Dyson NJ (2012) A shared role for RBF1 and dCAP-D3 in the regulation of transcription with consequences for innate immunity. PLoS Genetics 8(4):e1002618.
Popova MK, Korenjak M, He W, Dyson NJ, and Moon NS (2011) Rb deficiency during Drosophilaeye development deregulates EMC, causing defects in the development of photoreceptors and cone cells. Journal of Cell Science 124(Pt 24):4203-12.
Hsieh TC, Nicolay B, Frolov M, Moon NS (2010) Tuberous Sclerosis Complex 1 regulates dE2F1 expression during development and cooperates with RBF1 to control proliferation and survival. PLoS Genetics 6(8): e1001071 (Feature image for the August 2010 issue)
Morris EJ, Yuan JY, Yang F, Di Stefano L, Herr A, Moon NS, Kwon EJ, Haigis KM, Näär AM and Dyson NJ. (2008) E2F1 represses ß-catenin transcription and is antagonized by both pRB and CDK8. Nature. 455 (7212):552-6.
Moon NS, Di Stefano L, Morris EJ, Patel R, White K, and Dyson NJ (2008) E2F and p53 induce apoptosis independently during Drosophila development but intersect in the context of DNA damage. PLoS Genetics 4(8):e1000153.
Moon NS, Di Stefano L, and Dyson NJ (2006) A gradient of EGFR signaling determines the sensitivity of rbf1 mutant cells to E2F-dependent apoptosis. Molecular and Cellular Biology 26(20):7601-15.
Moon NS, Frolov MV, Kwon EJ, Di Stefano L, Dimova DK, Morris EJ, Taylor-Harding B, White K, Dyson NJ (2005) Drosophila E2F1 has context-specific pro- and antiapoptotic properties during development. Developmental Cell 9(4):463-75.
Frolov MV, Stevaux O, Moon NS, Dimova D, Kwon EJ, Morris EJ and Dyson NJ. (2003) G1 cyclin dependent kinases are insufficient to reverse dE2F2-mediated repression. Genes and Development17(6):723-728.
Kim M, Tang JP, Moon NS. (2018) An alternatively spliced form affecting the Marked Box domain of Drosophila E2F1 is required for proper cell cycle regulation. 59th Annual Drosophila Research Conference, Philadelphia, PA, USA
Bradley-Gill MR, Kim M, Moon NS (2018) Tsc/Tor pathway regulates specific de2f1 transcript-isoforms to control cell size and proper development. 59th Annual Drosophila Research Conference, Philadelphia, PA, USA
Kim M and Moon NS. (2017) Revealing complexities in the Drosophila RB/E2F pathway. 14th Canadian Drosophila Research Conference. Banff, AB, Canada.
Bradley-Gill MR, Kim M, Feingold D, Yergeau C, Moon NS (2016) Activator E2F is
needed for proper cell cycle exit during development. International RB meeting, Columbus, OH, USA, 2016
Collins H and Moon NS. (2015) dCAF1 is required for the cell death sensitivity of rbf1 mutant cells. 51st Annual Drosophila Research Conference, Chicago, Il, USA
Bradley-Gill MR and Moon NS. (2015) Identification of the specific de2f1 transcript that is required for the formation of imaginal discs. 51st Annual Drosophila Research Conference, Chicago, Il, USA
Collins H and Moon NS. The genetic interaction between rbf1 and Posterior sex combs reveals a role for histone chaperone dCAF-1 in cell survival (2012) Mechanism of Eukaryotic Transcription, Cold Spring Harbor, USA
Krivy K, Bradley-Gill MR, Moon NS. Capicua mutations promote proliferation and survival of Rb deficient cells in Drosophila. (2012) Society for Developmental Biology 71st annual meeting, Montreal, Canada